小儿在胎，其母脏气有热，熏蒸于胎，到生下小儿体皆黄，谓之胎疸也。
疟病者，由夏伤于暑，客于皮肤，至秋因劳动血气，腠理虚而邪乘之，动前暑热，正
邪相击，阴阳交争，阳盛则热，阴盛则寒。阴阳更盛更虚，故发寒热；阴阳相离，则寒热俱
歇。若邪动气至，交争复发，故疟休作有时。
其发时节渐晏者，此由邪客于风府，循膂而下，卫气一日一夜常大会于风府，其明日日
下一节，故其作日晏。其发早者，卫气之行风府，日下一节，二十一日下至尾 ，二十二日
入脊内，上注于伏冲之脉，其气上行九日，出于缺盆之内，其气既上，故其病发更早。
其间日发者，由邪气内薄五脏，横连募原，其道远，其气深，其行迟，不能日作，故间
日蓄积乃发也。
小儿未能触冒于暑，而亦病疟者，是乳母抱持解脱，不避风者也。
夫风邪所伤，是客于皮肤，而痰饮渍于脏腑，致令血气不和，阴阳交争。若真气胜，则
邪气退。邪气未尽，故发疟也。邪气虽退，气血上虚，邪气干于真气，脏腑壅痞，热气未散
，故余热往来也。
疟是夏伤于暑，热客于皮肤，至秋复为风邪所折，阴阳交争，故发寒热。其病正发，寒
热交争之时，热气乘脏，脏则燥而渴，渴而引饮，饮停成癖，结于胁下，故瘥后胁内结硬也
小儿在母胎中时，如果母亲的脏腑之气有热，热气熏蒸胎儿，等到孩子出生后，全身皮肤都会发黄，这被称为胎疸。
疟疾的发生，是因为夏季感受了暑邪，邪气潜伏在皮肤之间，到了秋季，因为劳累导致气血运行紊乱，腠理（皮肤纹理及汗孔）空虚，邪气趁机侵入，引动之前潜伏的暑热，正气与邪气相互搏击，阴阳发生交争。阳气偏盛则表现为发热，阴气偏盛则表现为恶寒。阴阳之气交替盛衰、虚实变化，所以出现寒热往来的症状；当阴阳暂时分离不相交争时，寒热症状就会停止。如果邪气再次触动正气，交争重新发作，所以疟疾的发作和休止都有一定的时间规律。
如果发作时间逐渐推迟，这是因为邪气客居在风府穴，沿着脊柱向下移动。卫气一天一夜常在风府穴大会，第二天卫气下行一节，所以发作日期逐渐推后。如果发作时间逐渐提前，是因为卫气行于风府，每天下行一节，二十一天下行至尾骶骨，第二十二日进入脊内，向上注入伏冲脉，卫气上行九天，出于缺盆穴内部。既然卫气已经上行，所以疾病发作得更早。
如果是隔日发作一次，是因为邪气向内迫近五脏，横向连接募原（脏腑之间的膜原），病位较远，邪气较深，运行缓慢，不能每天都发作，所以需要积蓄力量到一定程度才发作。
小儿没有直接触犯暑邪，却患上疟疾，这是因为乳母怀抱或照顾婴儿时疏忽大意，没有避开风邪所致。
凡是风邪所伤，邪气客居在皮肤，而痰饮浸渍于脏腑，导致气血不和，阴阳交争。如果人体真气强盛，则邪气退去。若邪气未完全清除，所以会引发疟疾。虽然邪气已退，但气血上部虚弱，邪气干扰真气，导致脏腑壅滞痞塞，热气未散，所以会有余热往来。
疟疾是夏季感受暑邪，热邪客居在皮肤，到了秋季又被风邪遏制，导致阴阳交争，所以出现寒热往来。在疾病正发、寒热交争的时候，热气乘虚侵犯内脏，内脏受热则干燥而口渴，口渴而饮水，水饮停聚形成癖积，结聚在胁下，所以在病愈之后，胁部内部仍有结块硬痛的感觉。
When a fetus is in the womb, if the mother's Zang-Fu Qi has heat, this heat steams and influences the fetus. Upon birth, the infant's entire body turns yellow, which is called "Tai Dan" (Fetal Jaundice).
Malaria occurs because summer-heat pathogen was injured in summer, lodging in the skin. By autumn, due to labor and exertion disrupting Qi and Blood, the Cou Li (skin pores and interstices) become deficient, allowing the pathogen to invade. This stirs up the previously latent summer-heat; Zheng Qi (Upright Qi) and Xie Qi (Pathogenic Qi) clash, causing Yin and Yang to contend. When Yang is excessive, there is fever; when Yin is excessive, there is chills. As Yin and Yang alternate in excess and deficiency, chills and fever occur; when Yin and Yang separate, both chills and fever cease. If the pathogen moves and Qi arrives, reigniting the contention, malaria recurs at regular intervals.
If the onset time gradually delays, it is because the pathogen lodges in Fengfu (GV16), moving down along the Du Meridian. Wei Qi (Defensive Qi) gathers at Fengfu once every day and night. The next day, it descends one vertebral level, so the onset date is delayed. If the onset time advances, it is because Wei Qi travels from Fengfu, descending one level daily. After twenty-one days, it reaches the coccyx; on the twenty-second day, it enters the spine, ascending into the Fu Chong Mai (Deep Rushing Vessel). It ascends for nine days, emerging within the Quepen area (supraclavicular fossa). Since Wei Qi has ascended, the disease manifests earlier.
If the fever occurs every other day, it is because the pathogen presses inward toward the Five Zang Organs and horizontally connects to the Mu Yuan (Sea of Origin/Membranous Origin). The pathway is distant, the Qi is deep, and its movement is slow, preventing daily occurrence. Thus, it accumulates until sufficient to manifest every other day.
Infants who have not directly exposed themselves to summer-heat but still contract malaria do so because their nursing mothers, while holding or caring for them, fail to avoid wind pathogens.
When injured by wind pathogen, the pathogen lodges in the skin, while phlegm-fluids soak into the Zang-Fu organs, causing disharmony of Qi and Blood and contention between Yin and Yang. If True Qi prevails, the pathogen retreats. If the pathogen is not fully cleared, malaria ensues. Although the pathogen may retreat, the upper part of Qi and Blood becomes deficient. The pathogen harasses True Qi, causing stagnation and obstruction in the Zang-Fu organs. With residual heat not yet dispersed, lingering heat causes alternating sensations.
Malaria results from summer-heat injury in summer, with heat lodging in the skin. In autumn, this is constrained by wind pathogen, leading to Yin-Yang contention and chills/fever. During the active phase, when chills and fever contend, heat attacks the Zang organs. The Zang organs become dry and thirsty. Thirst leads to drinking; stagnant fluid forms Pi accumulation (masses/lumps), knotting under the hypochondrium. Hence, after recovery, hardness remains internally within the hypochondriac region.